Search results for "Promoter activity"

showing 3 items of 3 documents

Regulation of the Expression of Inducible Nitric Oxide Synthase

2010

Publisher Summary This chapter reveals how nitric oxide (NO) generated by the inducible isoform of nitric oxide synthase (iNOS) exerts multiple beneficial microbicidal, antiviral, antiparasital, complex immunomodulatory, and antitumoral effects. Aberrant iNOS induction in the wrong place or at the wrong time has detrimental consequences and it is involved in the pathophysiology of several human diseases. Therefore, iNOS has to be regulated very tightly. The inducible isoform of NOS is mainly regulated at the level of expression. The mechanisms regulating iNOS expression involve modulation of promoter activity, mRNA stability and translatability, and protein stability. Modulation of iNOS exp…

Nitric oxide synthaseGene isoformMessenger RNAchemistry.chemical_compoundbiologychemistryPromoter activitybiology.proteinRNA-binding proteinTranscription factorPathophysiologyNitric oxideCell biology
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DNA damage response at telomeres boosts the transcription of SARS-CoV-2 receptor ACE2 during aging

2021

The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes the coronavirus disease 2019 (COVID-19), known to be more common in the elderly, who also show more severe symptoms and are at higher risk of hospitalization and death. Here, we show that the expression of the angiotensin converting enzyme 2 (ACE2), the SARS-CoV-2 cell receptor, increases during aging in mouse and human lungs. ACE2 expression increases upon telomere shortening or dysfunction in both cultured mammalian cells and in vivo in mice. This increase is controlled at the transcriptional level, and Ace2 promoter activity is DNA damage response (DDR)-dependent. Both pharmacological global DDR inhibition of ATM kin…

ace2; covid-19; dna damage response; aging; telomere; aged; angiotensin-converting enzyme 2; animals; humans; mice; sars-cov-2; aging; covid-19; dna damage; telomeremiceCoronavirus disease 2019 (COVID-19)DNA damageSevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)BiologySettore MED/08 - Anatomia PatologicaBiochemistry03 medical and health sciences0302 clinical medicineDownregulation and upregulationPromoter activityTranscription (biology)angiotensin-converting enzyme 2GeneticsSettore MED/05 - Patologia ClinicaReceptorhumansMolecular Biology030304 developmental biology0303 health sciencestelomereAce2 aging COVID-19DNA damage response telomereagingace23. Good healthTelomereCell biologybody regionsdna damage responseanimalsagedsars-cov-2covid-19Angiotensin-converting enzyme 2Cancer researchdna damagehormones hormone substitutes and hormone antagonists030217 neurology & neurosurgery
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Retinoids as a Perspective in Treatment of Alzheimer’s Disease

2010

<i>Background:</i> In the past, we demonstrated that the disintegrin metalloproteinase ADAM10 has α-secretase activity in vitro and in cultured cells. We also found out that moderate overexpression of this proteinase inhibits Aβ peptide production and prevents the formation of amyloid plaques in an Alzheimer’s disease (AD) mouse model. Moreover, it corrects early hippocampal defects like LTP impairment and increases cortical synaptogenesis. <i>Objective:</i> Upregulation of ADAM10 might be an alternative approach concerning AD therapy. Our current research therefore focuses on substances and/or pathways which regulate ADAM10 gene expression. <i>Methods:</i&g…

endocrine systemMorpholinesADAM10DiseaseBiologyADAM10 ProteinMiceNeuroblastomaRetinoidsPromoter activityCell Line TumorDisintegrinAnimalsHumansEnzyme InhibitorsMetalloproteinaseDose-Response Relationship DrugTerpenesPerspective (graphical)Membrane ProteinsVitaminshumanitiesIn vitroUp-Regulationcarbohydrates (lipids)ADAM ProteinsNeurologyChromonesImmunologyCancer researchbiology.proteinNeurology (clinical)Amyloid Precursor Protein SecretasesSignal TransductionNeurodegenerative Diseases
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